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Hori (1968) reported that E. rugosum gradually altered its feeding site from the vascular element to the mesophyll of the host plant with the advance of the bug stage. When the bug fed on the vascular element, there were no symptoms which could be observed externally, but the tube-like salivary sheath was formed along the whole length of the feeding puncture. When the bug feeds on the mesophyll, a unique small whitish spot appears on the plant, with an assemblage of radial or branch-like stripes starting from its centre. The size increases with the advance of the bug stage. These spots are made by the stylets which pass through the tissue and destroy the cells in their passage; injury is primarily due to mechanical destruction of cells and the withdrawal of the cell contents, but seems not to be caused by toxins in the salivary secretion. A young leaf under concentrated attack by the bugs withers very soon. Therefore when infestation by the bugs is severe in the seedling stage of cruciferous crops, damage is serious. In Japan, however, E. rugosum and E. pulchrum are not of very great economic importance.
Hori (1974, 1975) showed that the amounts of chlorophyll, amino acid and the activity of some oxidative enzymes in leaf tissue are changed by the feeding activity of the bugs. He suggested that these changes may halt the development of plant injury at the simple lesion stage.
Although no agricultural chemical pesticides for Eurydema have been registered in Japan, synthetic pyrethroids (cypermethrin, permethrin, tau-fluvalinate, bifenthrin, cyhalothrin, etofenprox and fenitrothion) could be used for the control of Eurydema. These pesticides are registered for stink bugs on other crops, and for other insects affecting crucifers. However, natural control by parasitoid wasps may be a preferable means of control.
In Japan, the economic impact of E. rugosum and E. pulchrum is very small. No agricultural chemical pesticides have been registered in Japan specifically for these stink bugs on crucifers.